Foville's syndrome

Category: Neuroanatomy.

When one half of the pons is injured involving the corticospinal tract (above the decussation of the pyramids), the facial nerve (CN VII) nucleus and/or facial nerve fibers, and the nucleus of CN VI (abducens) or the nearby paramedian pontine reticular formation (PPRF) fibers to CN VI, the resulting constellation of signs/symptoms includes: contralateral spastic weakness/paralysis (weakness, hypertonia, hyperreflexia and Babinski's sign), ipsilateral upper and lower facial weakness and loss of volitional abduction of the ipsilateral eye (horizontal gaze paresis).

From: http://en.wikipedia.org/wiki/Foville's_syndrome
Foville's syndrome is caused by the blockage of the perforating branches of the basilar artery in the region of the brainstem known as the pons.[1]
Structures affected by the infarct are the PPRF, nuclei of cranial nerves VI and VII, corticospinal tract, medial lemniscus, and the medial longitudinal fasciculus.

The syndrome is reported in http://www.nzma.org.nz/journal/119-1232/1928/ in which the case mentioned the syndrome on a suspected Wernicke’s encephalopathy.

There is another recorded case: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1143046/